Please use this identifier to cite or link to this item: http://dr.iiserpune.ac.in:8080/xmlui/handle/123456789/1394
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dc.contributor.authorMeena, H.en_US
dc.contributor.authorNakhate, Kartik T.en_US
dc.contributor.authorKokare, Dadasaheb M.en_US
dc.contributor.authorSUBHEDAR, NISHIKANT K.en_US
dc.date.accessioned2018-12-06T09:16:34Z
dc.date.available2018-12-06T09:16:34Z
dc.date.issued2009-01en_US
dc.identifier.citationLife Sciences, 84(5-6.).en_US
dc.identifier.issn0024-3205en_US
dc.identifier.urihttp://dr.iiserpune.ac.in:8080/xmlui/handle/123456789/1394-
dc.identifier.urihttps://doi.org/10.1016/j.lfs.2008.11.013en_US
dc.description.abstractWeight gain is a common outcome of antipsychotics therapy in schizophrenic patients. However, the underlying neuronal mechanisms are unclear. The present study was undertaken to investigate the role of GABAA receptors within the framework of nucleus accumbens shell (AcbSh) in haloperidol-induced hyperphagia and body weight gain in sated rats.en_US
dc.language.isoenen_US
dc.publisherElsevier B.V.en_US
dc.subjectAntipsychoticsen_US
dc.subjectNucleus accumbens shellen_US
dc.subjectFood intakeen_US
dc.subjectGABAA receptorsen_US
dc.subject2009en_US
dc.titleGABAA receptors in nucleus accumbens shell mediate the hyperphagia and weight gain following haloperidol treatment in ratsen_US
dc.typeArticleen_US
dc.contributor.departmentDept. of Biologyen_US
dc.identifier.sourcetitleLife Sciencesen_US
dc.publication.originofpublisherForeignen_US
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