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  • Phenotypic Plasticity and Effects of Selection on Cell Division Symmetry in Escherichia coli 
    LELE, UTTARA; BAIG, ULFAT; WATVE, MILIND (Public Library Science, 2011-01)
    Aging has been demonstrated in unicellular organisms and is presumably due to asymmetric distribution of damaged proteins and other components during cell division. Whether the asymmetry-induced aging is inevitable or an ...
  • Can Thrifty Gene(s) or Predictive Fetal Programming for Thriftiness Lead to Obesity? 
    BAIG, ULFAT; Belsare, Prajakta; WATVE, MILIND; Jog, Maithili (Hindawi, 2011-02)
    Obesity and related disorders are thought to have their roots in metabolic “thriftiness” that evolved to combat periodic starvation. The association of low birth weight with obesity in later life caused a shift in the ...
  • Protein Aggregation in E-coli: Short Term and Long Term Effects of Nutrient Density 
    BAIG, ULFAT; Bhadbhade, Bharati J.; MARIYAM, DINCY; WATVE, MILIND (Public Library Science, 2014-09)
    During exponential growth some cells of E. coli undergo senescence mediated by asymmetric segregation of damaged components, particularly protein aggregates. We showed previously that functional cell division asymmetry in ...
  • Evolution of Aging and Death: What Insights Bacteria Can Provide 
    BAIG, ULFAT; Bhadbhade, Bharati J.; WATVE, MILIND (The University of Chicago Press, 2014-09)
    Several unresolved issues, paradoxes, and information voids characterize the field of evolution of aging. The recent discovery of aging-like phenomenon in Escherichia coli, marked by asymmetric segregation of damaged ...
  • Cumulative cell division time asymmetry in Staphyloccus aureus 
    BAIG, ULFAT; SUNNY, RON; WATVE, MILIND; LELE, UTTARA (Science Matters AG, 2016-03)
    Asymmetry of cell division involving asymmetric damage segregation is shown to be linked to cellular senescence in bacteria. According to the current mainstream thinking, the asymmetry is observed along the old and new ...
  • The Evolving Opportunistic Pathogen Communities on Host Individuals and the Evolution of Host Aging 
    BAIG, ULFAT; Ojha, Akanksha; WATVE, MILIND (Asdin Publishing, 2017-04)
    Abstract In the coevolution of host and the associated opportunistically pathogenic microbiota, the microbiota has an advantage of a smaller generation time and thereby faster evolution. Sexual reproduction by the host is ...
  • Polarity, asymmetry and aging: are there Yayatis among bacteria? 
    BAIG, ULFAT; WATVE, MILIND; LELE, UTTARA (Indian Academy of Sciences, 2017-08)
    Bacteria have been shown to age. In an exponentially growing population some cells progressively slow down and stop dividing1 . This is thought to be due to asymmetric damage segregation in which old pole cells retain ...
  • Foraging theory and the propensity to be obese: an alternative to thrift 
    BAIG, ULFAT; LOKHANDE, LAVANYA; LALWANI, POORTATA; CHAWLA, SURAJ; Watve, Milind (Schweizerbart Science Publishers, 2019-11)
    The evolutionary origin of obesity is classically believed to be genetic or developmentally induced thrift, as an adaptation to ancestral feast and famine conditions. However, recently the thrift family of hypotheses have ...
  • Geriatric infections: Decreased immunity or evolved opportunists? 
    BAIG, ULFAT; LAXMI, VIDHYA; OJHA, AKANKSHA; Watve, Milind (Indian Academy of Sciences, 2020-04)
    In host–parasite co-evolution, parasites are assumed to have an advantage owing to their shorter generation time. Evolution of pathogens within the lifetime of a host individual is implicated as a strong selective force ...

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